| These observations established causality between the KCC3 T991A mutation and the neuropathy phenotype of the unique human patient, but also underscored species differences, as one copy of mutant was enough to produce the phenotype in a human patient, whereas two copies were necessary in mice <a href=http://clomid.buzz>clomid</a> This study clearly demonstrates that there are different prognostic subgroups of desmoid tumors that could benefit from different therapeutic strategies, including a wait and see policy |
|
